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The latter could be the result of PI3K/AKT signaling deregulation with the predominant disease-causing gene being PIK3R1. Our information claim that the phenotypic spectrum involving pathogenic variations of PTPN11 could possibly be broader than formerly explained, and also this could be as a result of the twin activity of SHP2 (ie, PTPN11 gene product) in the RAS/MAPK and PI3K/AKT signaling. This short article is shielded by copyright laws. All rights set aside. This article is shielded by