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Increased ST was significantly associated with developmental delay, in particular, in the domains of language acquisition and communication. In children aged ≥ 2 years, a delay in ≥ 3 domains was associated with ST (adjusted odds ratio [AOR] = 17.75, 95% CI 5.04-62.49, p less then 0.001), as was language delay (AOR = 52.92, 95% CI 12.33-227.21, p less then 0.001). In children aged less then 2 years, a delay in ≥ 2 domains was associated with ST (AOR = 16.79, 95% CI 2.26-124.4, p less then 0.001), as was language delay (AOR = 20.93, 95% CI

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05). Additionally, ASH1L knockdown inhibited cell proliferation, increased the apoptosis rate, and upregulated the expression of apoptosis genes CASPASE-3, BAX and BAX/BCL-2 ratio (P less then 0.05). Meanwhile, the mRNA expression levels of EZH2 and SUZ12, two subunits of PRC2 protein, were increased in cells with ASH1L knockdown (P less then 0.05). Therefore, the expression of ASH1L methyltransferase and its function in on the apoptosis of bovine cumulus cells were first studied. The mechanism by which ASH1L regulates the