https://pdesignaling.com/index.....php/antitussive-ant 
  Examining the underlined illness initiating mechanisms may well contribute to the development of book preventive therapies. Our aim would be to construct opn null mice in a NOD autoimmune-prone genetic history and address the pathogenic or protective role associated with osteopontin molecule during the early stages of kind 1 diabetes. Methods We generated opn null mutant mice in a NOD genetic back ground by serial backcrossing into the present C57BL/6 opn knoc
 
					 
						 
                    
                                     
       
       
       
       
       
       
       
       
       
         