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Dexmedetomidine post-conditioning attenuates cerebral ischemia subsequent asphyxia stroke via down-regulation associated with apoptosis as well as neuroinflammation throughout subjects. The inclusion of sodium chloride in the pellet increased the release rate and reduced initial lag. The release was sustained for 100 days. CONCLUSIONS The release rate of tenofovir alafenamide mechanistically varied with geometry and rate controlling membrane composition. The polyether urethane implant presented